机译:5-氮杂胞苷和NF-KB抑制剂DHMEQ对髓性白血病细胞凋亡诱导的协同作用
Department of Hematology School of Medicine Kitasato University 1-15-1 Kitasato Minami-ku;
Department of Hematology School of Medicine Kitasato University 1-15-1 Kitasato Minami-ku;
Department of Hematology School of Medicine Kitasato University 1-15-1 Kitasato Minami-ku;
School of Medicine Aichi Medical University Nagakute Aichi Japan;
Laboratory of Tumor Cell Biology Department of Medical Genome Sciences Graduate School of;
Department of Hematology School of Medicine Kitasato University 1-15-1 Kitasato Minami-ku;
Department of Hematology School of Medicine Kitasato University 1-15-1 Kitasato Minami-ku;
5-Azacytidine (AZA); Dehydroxymethylepoxyquinomicin (DHMEQ); Leukemia; NF-kB;
机译:5-氮胞苷和NF-kB抑制剂DHMEQ对髓样白血病细胞凋亡诱导的协同作用
机译:MicroRNA-125B的过度表达抑制人急性髓性白血病细胞侵袭,增殖和促进细胞凋亡,靶向NF-KB信号通路
机译:DHMEQ是一种新型的NF-kB抑制剂,可诱导人肝癌细胞的凋亡和细胞周期停滞
机译:降解的胶原片段激活NF-KB并通过诱导凋亡蛋白抑制剂(圈)保护人平滑肌细胞免受细胞凋亡
机译:ALK和FLT3激酶抑制剂对非小细胞肺癌细胞和急性髓系白血病细胞的癌症驱动突变的药理作用
机译:4-羟肉桂酸甲酯和肌酸通过钙依赖性细胞凋亡诱导对急性髓样白血病细胞的协同细胞毒性
机译:XPO1和FLT3的组合靶向通过在FLT3-突变的急性髓性白血病中的分化和细胞凋亡诱导协同抗白血病效应:从概念到临床试验