机译:DL-3-N-丁基苯乙烯(NBP)在通过NRF2的创伤性脑损伤后在小鼠模型中提供神经保护作用 - 信号传导途径
Southern Med Univ Guangzhou Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing;
Southern Med Univ Guangzhou Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing;
Shenzhen Longgang Cent Hosp Dept Neurosurg 6082 Longgangdadao Rd Shenzhen 518116 Guangdong;
Nanjing Univ Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing 210002 Jiangsu;
Nanjing Univ Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing 210002 Jiangsu;
Southern Med Univ Guangzhou Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing;
Southern Med Univ Guangzhou Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing;
Nanjing Univ Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing 210002 Jiangsu;
Nanjing Univ Sch Med Jinling Hosp Dept Neurosurg 305 East Zhongshan Rd Nanjing 210002 Jiangsu;
DL-3-n-Butylphthalide; Neuroprotection; Traumatic brain injury; Nrf2-ARE signaling pathway; Mice model;
机译:DL-3-N-丁基苯乙烯(NBP)在通过NRF2的创伤性脑损伤后在小鼠模型中提供神经保护作用 - 信号传导途径
机译:姜黄素通过NRF2的创伤性脑损伤模型提供神经保护作用 - 是信号通路
机译:褪黑素通过激活Nrf2-ARE信号通路在小鼠颅脑外伤模型中调节抗氧化酶的活性
机译:6.4:演讲环节:神经解剖学,神经再生和建模:“脑外伤后的再生:信号,路标和支架”
机译:脑外伤后NR2B和自噬信号通路的激活
机译:岩藻黄质通过Nrf2-ARE和Nrf2自噬途径在颅脑外伤模型中提供神经保护作用
机译:N-乙酰半胱氨酸酰胺通过NRF2提供神经保护作用的神经口腔脑损伤的小鼠模型中的途径