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Episodic creep events on the San Andreas Fault caused by pore pressure variations

机译:由孔隙压力变化引起的San Andreas故障的episodic蠕变事件

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Recent seismic and geodetic observations indicate that interseismic creep rate varies in both time and space. The spatial extent of creep pinpoints locked asperities, while its temporary accelerations, known as slow-slip events, may trigger earthquakes. Although the conditions promoting fault creep are well-studied, the mechanisms for initiating episodic slow-slip events are enigmatic. Here we investigate surface deformation measured by radar interferometry along the central San Andreas Fault between 2003 and 2010 to constrain the temporal evolution of creep. We show that slow-slip events are ensembles of localized creep bursts that aseismically rupture isolated fault compartments. Using a rate-and-state friction model, we show that effective normal stress is temporally variable on the fault, and support this using seismic observations. We propose that compaction-driven elevated pore fluid pressure in the hydraulically isolated fault zone and subsequent frictional dilation cause the observed slow-slip episodes. We further suggest that the 2004 M-w 6 Parkfield earthquake might have been triggered by a slow-slip event, which increased the Coulomb failure stress by up to 0.45 bar per year. This implies that while creeping segments are suggested to act as seismic rupture barriers, slow-slip events on these zones might promote seismicity on adjacent locked segments.
机译:最近的地震和大地测量观测表明,在时间和空间中造型蠕变率变化。蠕变精确点的空间程度锁定粗糙度,而其临时加速度,称为慢速滑移事件,可能会触发地震。虽然促进了故障蠕变的条件很好地研究,但启动情节速率慢速事件的机制是神秘的。在这里,我们研究了2003年至2010年之间的雷达干涉测量测量的表面变形,以限制蠕变的时间演变。我们表明,慢速泄放事件是局部蠕变爆发的集成,即崩溃隔离故障隔室。使用速率和状态摩擦模型,我们表明,在故障上有效的正常压力是在时间上变化的,并使用地震观察来支持这种情况。我们提出压实驱动的升高的升高的孔隙流体压力和随后的摩擦扩张导致观察到的慢速发作。我们进一步建议,2004年M-W 6 Parkfield地震可能被慢滑事件引发,每年将库仑故障压力增加到0.45巴。这意味着,虽然建议蔓延段充当地震破裂障碍,但这些区域上的慢速泄放事件可能促进相邻的锁定段上的地震性。

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