机译:白细胞介素-33缺乏症加剧了实验性自身免疫性脑脊髓炎,影响免疫细胞和胶质细胞
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Clinical laboratory The First Affiliated Hospital of Hainan Medical University;
Department of Laboratory Medicine The Second Affiliated Hospital of Guilin Medical University;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Department of Immunology School of Basic Medicine Tongji Medical College Huazhong University of;
Astrocytes; EAE; Effector T cells; Interleukin-33; Microglia/macrophages; Regulatory T cells;
机译:白细胞介素-33缺乏症加剧了实验性自身免疫性脑脊髓炎,影响免疫细胞和胶质细胞
机译:Blimp-1缺乏通过损害Treg细胞的IL-10产生通过损害小鼠的实验自身免疫性脑脊髓炎
机译:树突状细胞的Mertk缺乏症加剧了实验性自身免疫性脑脊髓炎
机译:旋转磁场通过促进T细胞周围积累和调节Treg和Th1 / Th17的平衡来改善实验性自身免疫性脑炎。
机译:CD44缺乏症的多方面的作用有助于增强实验性自身免疫性脑脊髓炎和血管屏障功能障碍:在血管的免疫细胞,血脑屏障和内皮细胞中的作用。
机译:实验性自身免疫性脑脊髓炎期间Nogo-受体1缺乏对免疫细胞库或功能没有影响
机译:在实验性自身免疫性脑脊髓炎期间,Nogo-受体1缺乏对免疫细胞库或功能没有影响。