机译:瘦素通过KLF4和SOCS3抑制STAT3信号传导刺激海马神经元的Sysemation
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Oregon Hlth &
Sci Univ Oregon Stem Cell Ctr Portland OR 97239 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Washington State Univ Dept Integrated Physiol &
Neurosci Pullman WA 99164 USA;
Leptin; STAT3; KLF4; SOCS3; Dendritic spine; Hippocampus;
机译:瘦素通过KLF4和SOCS3抑制STAT3信号传导刺激海马神经元的Sysemation
机译:Leptin受体敲除诱导的抑郁型行为和锻炼的减毒抗抑郁作用与STAT3 / SOCS3信号传导相关
机译:过氧化物酶体增殖物激活的受体-β/-δ(PPAR-β/-δ)的激活通过抑制白介素6刺激的脂肪细胞中的STAT3改善胰岛素信号传导并降低SOCS3水平。
机译:Abeta通过在海马神经元中扰乱JAK2 / Stat3轴来引起内存损伤。
机译:海马神经元的微模式;表征和研究突触形成的意义
机译:独立于瘦素受体/ STAT3信号传导适当抑制排汗性下丘脑弓形核神经元
机译:由JAK STAT3和线粒体稳定介导的海马神经元中的瘦素介导的细胞存活信号