机译:pparγ激动剂在Alzheimer疾病TG2576小鼠模型中抢救增加FGF14的磷酸化
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Internal Medicine University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Neuroscience Graduate Program Graduate School of Biomedical Sciences University of Texas Medical;
Neuroscience Graduate Program Graduate School of Biomedical Sciences University of Texas Medical;
Biochemistry and Molecular Biology Graduate Program Graduate School of Biomedical Sciences;
Mitchell Center for Neurodegenerative Diseases University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Mitchell Center for Neurodegenerative Diseases University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Department of Internal Medicine University of Texas Medical Branch;
Department of Pharmacology &
Toxicology University of Texas Medical Branch;
Alzheimer's disease; PPARgamma; Fibroblast growth factor 14; Mass spectrometry; Confocal microscopy;
机译:pparγ激动剂在Alzheimer疾病TG2576小鼠模型中抢救增加FGF14的磷酸化
机译:在Alzheimer疾病的TG2576小鼠疾病的Hippocampus中早期破坏帕蒿蛋白表达和脑尿位的蚊帐可以通过丰富的环境来救出
机译:LXR激动剂TO901317在阿尔茨海默氏病的Tg2576小鼠模型中选择性降低海马A beta 42并改善记忆
机译:pParγ-和Toll样受体-2激动剂可能对呼吸道合胞病毒(RSV)的治疗值(RSV)诱导的气道疾病Pparγ激动剂可以以ppargamma独立的方式行事
机译:慢性交通噪声暴露导致认知和运动功能障碍的出现,并增加了阿尔茨海默氏病小鼠模型中的淀粉样蛋白-β沉积
机译:PPARγ激动剂可挽救阿尔茨海默氏病Tg2576小鼠模型中S226 FGF14的磷酸化
机译:泄漏的Ryanodine受体增加了Alzheimer病的Tg2576小鼠模型的β-淀粉样蛋白负荷并诱导记忆障碍