机译:IL IL -33诱导自身抗体呼吸上皮细胞的自身抗体的产生:COPD COPD发病机制的潜在机制
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of Laboratory Animal SciencesCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of Respiratory and Critical Care MedicineCapital Medical University &
Beijing Institute;
Faculty of Life Sciences &
MedicineAsthma UK Centre in Allergic Mechanisms of Asthma King's College;
Department of ImmunologyCapital Medical UniversityBeijing China;
Department of ImmunologyCapital Medical UniversityBeijing China;
alveolar type II epithelial cell; autoimmune; chronic obstructive pulmonary disease; emphysema; IL ‐33;
机译:IL IL -33诱导自身抗体呼吸上皮细胞的自身抗体的产生:COPD COPD发病机制的潜在机制
机译:可溶性ST2抑制上皮细胞衍生的IL-33诱导人嗜碱性KU812细胞产生IL-5
机译:COPD患者上皮细胞中响应香烟烟雾的IL-8产生减少
机译:miR223对牙龈上皮细胞IL-1β和IL-6产生的影响
机译:巨噬细胞集落刺激因子诱导人脐带血单核细胞分化为具有耐受原性的IL-10(高)IL-12(缺失)树突状细胞和稀有的CD16(+)单核细胞。
机译:IL-33诱导针对自体呼吸道上皮细胞的自身抗体的产生:COPD发病机理的潜在机制
机译:IL -33诱导自身抗体呼吸上皮细胞的自身抗体的产生:一种潜在机制,用于COPD的发病机制