机译:血管紧张素 - (1-7)对通过PI3K / AKT信号通路对H9C2心肌细胞细胞高血糖血症诱导损伤的保护作用
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
Guangzhou Med Univ Affiliated Hosp 2 Intens Care Unit Guangzhou 510260 Guangdong Peoples R;
Laiwu City Peoples Hosp Dept Cardiol Laiwu 27110 Shandong Peoples R China;
Sun Yat Sen Cardiovasc Hosp Dept Cardiol Shenzhen 518020 Guangdong Peoples R China;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiol 58 Zhongshan Second Rd Guangzhou 510080;
angiotensin-(1-7); high glucose; phosphoinositide 3-kinase and protein kinase B pathway; cardiomyocyte;
机译:血管紧张素-(1-7)通过PI3K?Akt信号通路对高糖诱导的H9c2心肌母细胞损伤的保护作用
机译:血管紧张素 - (1-7)对H9C2心肌细胞细胞高血糖血症损伤的保护作用通过PI3K / AKT信号通路(Vol 41,PG 1283,2018)
机译:[更正]血管紧张素?(1?7)通过PI3K?Akt信号通路对高糖血症诱导的H9c2?心肌细胞损伤的保护作用
机译:PI3K / AKT /GSK3β途径参与神经元和胶质细胞的光动力损伤
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:血管紧张素-(1-7)通过PI3K̸Akt信号通路对高血糖诱导的H9c2心肌母细胞损伤的保护作用
机译:Kirenol通过PI3K / AKT途径通过NRF2信号传导H9C2细胞对多柔比蛋白诱导的心脏肥大的心脏保护作用