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机译:通过Cilengitide可以预防人类克罗恩病患中αvF-β1的激活增加,Cilengitide可以预防TNBS结肠炎中的纤维化
Departments of Medicine Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Medicine Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Medicine Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Medicine Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Surgery Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Surgery Medical College of Virginia Campus Virginia Commonwealth University;
Departments of Medicine Medical College of Virginia Campus Virginia Commonwealth University;
Collagen; Proximity ligation assay; RGD domain; Smad3; TNBS colitis;
机译:西仑吉肽可预防αVβ3在人克罗恩病和TNBS结肠炎纤维化中增加潜在的TGF-β1活化
机译:增加了与Barrett食管炎,慢性胃炎,麸质萎缩十二肾炎(腹腔疾病),淋巴细胞结肠炎,胶原性结肠炎,溃疡性结肠炎和克罗恩的结肠炎相关的溶菌酶产生的增加
机译:与细菌增殖相关的溶菌酶的生产增加与巴雷特食管炎,慢性胃炎,面筋诱发的萎缩性十二指肠炎(乳糜泻),淋巴细胞性结肠炎,胶原性结肠炎,溃疡性结肠炎和克罗恩氏结肠炎有关
机译:分析综合性炎症性肠病小鼠模型以评估其疾病驱动途径以及与克罗恩病和溃疡性结肠炎的相关性
机译:克罗恩病,溃疡性结肠炎和表型正常个体的表观遗传
机译:西仑吉肽可预防αVβ3在人克罗恩病和TNBS结肠炎纤维化中增加潜在的TGF-β1活化
机译:Cilengitiens可以预防人类克罗恩病患中αvβ3的患者蛋白疾病和纤维化的潜伏TGF-β1的激活