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机译:病毒特异性CD8 + T细胞的分化和保护能力推动小鼠诺罗病毒持续存在于免疫特异性肠道利基
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Medicine Division of Gastroenterology University of Pennsylvania Perelman School of;
Department of Microbiology University of Pennsylvania Perelman School of Medicine Philadelphia;
Department of Microbiology University of Pennsylvania Perelman School of Medicine Philadelphia;
Department of Microbiology and Immunology Thomas Jefferson University Philadelphia PA 19107 USA;
Department of Pathology and Laboratory Medicine Children’s Hospital of Philadelphia Research;
Department of Molecular Microbiology and Immunology Oregon Health and Science University Portland;
Department of Pathology and Immunology Washington University School of Medicine St. Louis MO;
Department of Microbiology University of Pennsylvania Perelman School of Medicine Philadelphia;
norovirus; enteric viral persistence; T cell inflation; T cell exhaustion; tissue-resident memory T?cells;
机译:病毒特异性CD8 + T细胞的分化和保护能力推动小鼠诺罗病毒持续存在于免疫特异性肠道利基
机译:对大量和病毒特异性CD8 + T细胞的分析显示,在具有常见可变免疫缺陷的患者中,CD8 + T细胞具有晚期分化。
机译:共生微生物和干扰素-λ确定肠鼠诺如病毒感染的持续性
机译:Tapak Liman(elephantopus scaber l)提取诱导的CD4 +和CD8 +与小鼠造血干细胞和祖细胞增殖的分化(Mus Musculus L)
机译:细胞本征转化生长因子-β信号传导介导病毒特异性CD8 + T细胞缺失和淋巴细胞脉络膜脑膜炎病毒在体内的持久性
机译:病毒特异性CD8 + T细胞的分化和保护能力表明鼠诺如病毒在免疫弱势肠壁中的持久性
机译:持续性肠道鼠诺如病毒感染与功能欠佳的病毒特异性CD8 T细胞反应有关