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机译:肝再生增强器的过度表达(ALR)减轻了H2O2诱导的内质网胁迫在肾小管上皮细胞中的作用
Chongqing Med Univ Affiliated Hosp 2 Dept Nephrol Chongqing 400010 Peoples R China;
Chongqing Med Univ Affiliated Hosp 2 Dept Nephrol Chongqing 400010 Peoples R China;
Chongqing Med Univ Affiliated Hosp 2 Dept Nephrol Chongqing 400010 Peoples R China;
Chongqing Med Univ Key Lab Mol Biol Infect Dis Inst Viral Hepatitis Dept Infect Dis Minist Educ Affiliated Hosp 2 Chongqing 400010 Peoples R China;
Chongqing Med Univ Key Lab Mol Biol Infect Dis Inst Viral Hepatitis Dept Infect Dis Minist Educ Affiliated Hosp 2 Chongqing 400010 Peoples R China;
Chongqing Med Univ Affiliated Hosp 2 Dept Nephrol Chongqing 400010 Peoples R China;
Augmenter of liver regeneration; Endoplasmic reticulum stress; Apoptosis; Acute kidney injury;
机译:肝再生增强器的过度表达(ALR)减轻了H2O2诱导的内质网胁迫在肾小管上皮细胞中的作用
机译:mapk磷酸酶-1上调对白蛋白诱导的肾小管细胞内质网应激的调节
机译:vaspin通过抑制HMGB1减少肾小管上皮细胞的炎症和内质网胁迫并降低肾缺血再灌注损伤
机译:生物肾上肾小管上皮细胞的二维细胞片状操纵
机译:小鼠肝细胞系中内质网伴侣伴侣调节剂的诱导型过表达。
机译:海藻糖通过减轻ROS依赖性内质网应激和AMPK激活来抑制H2O2诱导的多巴胺能SH-SY5Y细胞自噬死亡
机译:海藻糖通过减轻ROS依赖性内质网应激和AMPK激活,抑制多巴胺能SH-SY5Y细胞中的H2O2诱导的自噬死亡