...
机译:人类2型先天淋巴细胞破坏皮肤角膜炎狭窄的接线屏障IL-13
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Division of Dermatology Department of Medicine of Sensory and Motor Organs Faculty of;
Division of Dermatology Department of Medicine of Sensory and Motor Organs Faculty of;
Division of Dermatology Department of Medicine of Sensory and Motor Organs Faculty of;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
Swiss Institute of Allergy and Asthma Research (SIAF)University of ZurichDavos Switzerland;
机译:人类2型先天淋巴细胞破坏皮肤角膜炎狭窄的接线屏障IL-13
机译:前列腺素I2抑制人2型先天淋巴样细胞产生IL-33诱导的IL-5和IL-13
机译:IL-33在激发产生IL-13的核细胞(2型先天性淋巴样细胞)和气道收缩方面比IL-25更有效。
机译:守卫:先天淋巴细胞促进阻挡表面的免疫和组织稳态
机译:细胞对人类皮肤中促炎性细胞因子的反应:TNF-α对HDMEC中基因转录的调节,以及角质形成细胞对IL-18的激活和分泌
机译:前列腺素I2抑制人2型先天性淋巴样细胞产生IL-33诱导的IL-5和IL-13
机译:2型先天淋巴细胞通过在哮喘患者中通过IL-13靶向紧致连接来破坏支气管上皮阻挡层的完整性