机译:降低对乙醇的敏感性,并在神经病疼痛的小鼠模型中过度饮用
Institute of Psychopharmacology Central Institute of Mental Health Medical Faculty of;
Institute of Psychopharmacology Central Institute of Mental Health Medical Faculty of;
Institute of Psychopharmacology Central Institute of Mental Health Medical Faculty of;
Institute of Psychopharmacology Central Institute of Mental Health Medical Faculty of;
Neurobiology of Behavior Research Group (GReNeC‐NeuroBio) Department of Health and Experimental;
Institute of Psychopharmacology Central Institute of Mental Health Medical Faculty of;
alcohol drinking; chronic pain; intoxication; negative affect; pain thresholds; relapse; sex‐specific alcohol effects;
机译:降低对乙醇的敏感性,并在神经病疼痛的小鼠模型中过度饮用
机译:在炎性和神经性三叉神经痛小鼠模型中评估伤害感受敏感性。
机译:活性氧和脂质过氧化抑制剂会降低大鼠脊髓损伤的慢性神经性疼痛模型的机械敏感性
机译:自发性疼痛的临床前替代模型:神经性疼痛中药物发育的机会
机译:电压门控钙通道亚基α2delta-1和血小板反应蛋白-4相互作用引起的适应性突触形成是神经损伤模型中神经性疼痛处理的新机制。
机译:活性氧和脂质过氧化抑制剂会降低大鼠脊髓损伤的慢性神经性疼痛模型的机械敏感性
机译:一种新型的Mu-delta阿片类激动剂表明,在小鼠神经性疼痛模型中降低了耐受性和依赖性的增强效力