Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson's disease pathogenesis (vol 137, pg 103, 2019)

Hughes Craig D.; Choi Minee L.; Ryten Mina; Hopkins Lee; Drews Anna; Botia Juan A.; Iljina Maria; Rodrigues Margarida; Gagliano Sarah A.; Gandhi Sonia; Bryant Clare; Klenerman David

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Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson's disease pathogenesis (vol 137, pg 103, 2019)

机译：寡聚α-突触核蛋白的皮摩尔浓度敏感TLR4在帕金森病发病机制中发挥起始作用（第137 Vol 137，PG 103,2019）

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In the original publication of this article, the author Magarida Rodrigues was written incorrectly.

机译：在本文的原始出版物中，作者Magarida RodRigues被错误地写作。

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《Acta Neuropathologica 》 |2019年第1期| 共1页
作者
Hughes Craig D.; Choi Minee L.; Ryten Mina; Hopkins Lee; Drews Anna; Botia Juan A.; Iljina Maria; Rodrigues Margarida; Gagliano Sarah A.; Gandhi Sonia; Bryant Clare; Klenerman David;
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作者单位

Univ Cambridge Dept Vet Med Cambridge CB3 0ES England;

UCL Dept Clin &

Movement Neurosci Inst Neurol Queen Sq London WC1N 3BG England;

UCL Dept Mol Neurosci Inst Neurol London WC1N 3BG England;

Univ Cambridge Dept Vet Med Cambridge CB3 0ES England;

Univ Cambridge Dept Chem Cambridge CB2 1EW England;

UCL Dept Mol Neurosci Inst Neurol London WC1N 3BG England;

Univ Cambridge Dept Chem Cambridge CB2 1EW England;

Univ Cambridge Dept Chem Cambridge CB2 1EW England;

Univ Michigan Ctr Stat Genet Ann Arbor MI 48109 USA;

Francis Crick Inst 1 Midland Rd London NW1 1AT England;

Univ Cambridge Dept Vet Med Cambridge CB3 0ES England;

Univ Cambridge Dept Chem Cambridge CB2 1EW England;

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正文语种 eng
中图分类神经病学与精神病学 ;
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1. Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson's disease pathogenesis (vol 137, pg 103, 2019) [J] . Hughes Craig D., Choi Minee L., Ryten Mina, Acta Neuropathologica . 2019 ,第1期

机译：寡聚α-突触核蛋白的皮摩尔浓度敏感TLR4在帕金森病发病机制中发挥起始作用（第137 Vol 137，PG 103,2019）
2. alpha-Synuclein up-regulates expression of caveolin-1 and down-regulates extracellular signal-regulated kinase activity in B103 neuroblastoma cells: role in the pathogenesis of Parkinson's disease. [J] . Hashimoto M, Takenouchi T, Rockenstein E, Journal of Neurochemistry: Offical Journal of the International Society for Neurochemistry . 2003 ,第6期

机译：α-突触核蛋白上调B103神经母细胞瘤细胞中Caveolin-1的表达并下调细胞外信号调节的激酶活性：在帕金森氏病发病机理中的作用。
3. Toxic Oligomeric Alpha-Synuclein Variants Present in Human Parkinson’s Disease Brains Are Differentially Generated in Mammalian Cell Models [J] . Jie Wu, Michael R. Sierks, Now Bahar Alam, Biomolecules . 2015 ,第3期

机译：人类帕金森氏病大脑中存在的有毒寡聚α-突触核蛋白变体在哺乳动物细胞模型中差异产生
4. The relationship between aggregation of alpha-synuclein and the pathogenesis of Parkinson's disease. [D] . Wan, Oi Wan. 2011

机译：α-突触核蛋白的聚集与帕金森氏病发病机理之间的关系。
5. Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson’s disease pathogenesis [O] . Craig D. Hughes, Minee L. Choi, Mina Ryten, -1

机译：皮摩尔浓度的寡聚α-突触核蛋白使TLR4在帕金森氏病发病机理中起起始作用
6. CpG Demethylation Enhances Alpha-Synuclein Expression and Affects the Pathogenesis of Parkinson's Disease [O] . Matsumoto, Lumine, Takuma, Hiroshi, Tamaoka, Akira, 2010

机译：CpG脱甲基增强α-突触核蛋白表达并影响帕金森氏病的发病机理

Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson's disease pathogenesis (vol 137, pg 103, 2019)

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