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机译:增强的单核细胞募集和延迟替代巨噬细胞极化伴随着在C57BL / 6中的心肌梗死后的修复受损,与BALB / C小鼠相比
BHF/University Centre for Cardiovascular Science Queen’s Medical Research Institute University of;
Faculty of Biology Medicine and Health School of Biological SciencesUniversity of;
MRC Centre for Inflammation Research Queen’s Medical Research Institute University of;
BHF/University Centre for Cardiovascular Science Queen’s Medical Research Institute University of;
MRC Centre for Inflammation Research Queen’s Medical Research Institute University of;
BHF/University Centre for Cardiovascular Science Queen’s Medical Research Institute University of;
Faculty of Biology Medicine and Health School of Biological SciencesUniversity of;
BHF/University Centre for Cardiovascular Science Queen’s Medical Research Institute University of;
C57BL/6; BALB/c; macrophage polarization; monocyte; myocardial infarction;
机译:增强的单核细胞募集和延迟替代巨噬细胞极化伴随着在C57BL / 6中的心肌梗死后的修复受损,与BALB / C小鼠相比
机译:在无法治愈的BALB / c小鼠中,替代性巨噬细胞激活的障碍会延迟皮肤利什曼病的发生。
机译:替代性巨噬细胞激活的损害延迟了非愈合BALB / c小鼠皮肤利什曼病的发生。
机译:壳聚糖水凝胶可通过挽救ROS引起的细胞粘附受损,改善缺血性心肌微环境,提高干细胞的心肌修复效率。
机译:与BALB / c小鼠相比C57BL / 6心肌梗死后增强的单核细胞募集和延迟的替代性巨噬细胞极化伴随受损的修复
机译:增强的单核细胞募集和延迟替代巨噬细胞极化伴随着在C57BL / 6中的心肌梗死后的修复受损,与BALB / C小鼠相比