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机译:Kinin B1受体调节肌肉特异性E3连接酶的表达,并参与骨骼肌肿块控制
Department of Biochemistry and Immunology Faculty of Medicine at Ribeir?o Preto University of S?o;
Department of Biochemistry and Immunology Faculty of Medicine at Ribeir?o Preto University of S?o;
Department of Biochemistry and Immunology Faculty of Medicine at Ribeir?o Preto University of S?o;
Department of Pharmacology Escola Paulista de Medicina Universidade Federal de S?o Paulo 04039;
Department of Biophysics Escola Paulista de Medicina Universidade Federal de S?o Paulo 04039-032;
Department of Biochemistry and Immunology Faculty of Medicine at Ribeir?o Preto University of S?o;
Department of Pharmacology Escola Paulista de Medicina Universidade Federal de S?o Paulo 04039;
Department of Biochemistry and Immunology Faculty of Medicine at Ribeir?o Preto University of S?o;
G-protein-coupled receptor (GPCR); muscle atrophy; Protein metabolism; Signal transduction; Ubiquitin-proteasome system;
机译:激肽B1受体调节肌肉特异性E3连接酶的表达,并参与骨骼肌质量控制
机译:激肽B1受体调节肌肉特异性E3连接酶的表达,并参与骨骼肌质量控制
机译:Kinin B1受体调节肌肉特异性E3连接酶的表达,并参与骨骼肌肿块控制
机译:糖尿病迷你猪骨骼肌中ATP结合盒转运蛋白A1和肝X受体α表达的变化糖尿病骨骼肌LXRα/ ABCA1途径的变化
机译:骨骼肌中的肽酶调节血管紧张素,激肽和神经激肽的局部浓度。
机译:骨骼肌11β-HSD1控制糖皮质激素诱导的蛋白水解和E3泛素天冬氨酸Atrogin-1和MuRF-1的表达。
机译:E3泛素连接酶WWP1,与AMPKα2相互作用,下调其在骨骼肌C2C12细胞中的表达