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机译:BCLAF1对HDAC抑制剂LMK-235介导的弥漫性大B细胞淋巴瘤细胞凋亡的影响及其机制
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Dept Pharm Guiyang Guizhou Peoples R China;
Guizhou Med Univ Dept Pharm Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
Guizhou Med Univ Guiyang Guizhou Peoples R China;
LMK-235; BCLAF1; HDAC4; NF-kB; apoptosis; diffuse large B cell lymphoma;
机译:BCLAF1对HDAC抑制剂LMK-235介导的弥漫性大B细胞淋巴瘤细胞凋亡的影响及其机制
机译:I类HDAC抑制剂MGCD0103诱导霍奇金淋巴瘤细胞系凋亡,并通过不依赖HDAC6的机制与蛋白酶体抑制剂协同作用。
机译:I类HDAC抑制剂MGCD0103诱导霍奇金淋巴瘤细胞系凋亡,并通过不依赖HDAC6的机制与蛋白酶体抑制剂协同作用
机译:通过NF-к-b-β-抑制剂(Bay 11-7085)通过NF-к-B-B-B-B-B-B-β-β-抑制剂诱导P53缺陷L1210Cells的凋亡
机译:抗凋亡的新机制:细胞表面Galectin-3和CD45的相互作用促进弥漫性大B细胞淋巴瘤细胞的存活。
机译:BCLAF1对HDAC抑制剂LMK-235介导的弥漫性大B细胞淋巴瘤细胞凋亡的影响及其机制
机译:第二代抑菌剂卡非昔布与HDAC抑制剂协同作用,通过多种机制和环孢菌抗性扩散了大型B细胞淋巴瘤细胞