机译:K-Ras通过抑制非规范WNT信号传导来促进致瘤性
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
Univ Calif San Francisco Helen Diller Family Comprehens Canc Ctr San Francisco CA 94158 USA;
机译:K-Ras通过抑制非规范WNT信号传导来促进致瘤性
机译:非经典的wnt信号拮抗,经典的wnt信号促进肾脏早期发育中的细胞增殖。
机译:K-ras Asp12突变体既不与Raf相互作用,也不通过Erk发出信号,并且其致瘤性低于K-ras Val12
机译:粗糙度表面修饰调节HMCS中Wnt的规范和非规范信号通路
机译:通过ROR2和VANGL2在乳腺发育中的非经典WNT信号传导。
机译:非规范WNT信号拮抗和规范WNT信号促进早期肾发育中细胞增殖
机译:K-Ras通过抑制非经典Wnt信号传导促进致瘤性。