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机译:MiR-191被血小板衍生的微泡分泌诱导肾小管上皮细胞凋亡,并通过抑制CBS参与肾缺血再灌注损伤
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
Zhengzhou Univ Peoples Hosp Henan Prov Peoples Hosp Dept Urol Zhengzhou Henan Peoples R China;
miR-191; Cystathionine-beta-synthase; renal transplantation; renal ischemia-reperfusion injury;
机译:MiR-191被血小板衍生的微泡分泌诱导肾小管上皮细胞凋亡,并通过抑制CBS参与肾缺血再灌注损伤
机译:慢病毒介导的Src同源性2域蛋白酪氨酸磷酸酶2沉默通过抑制TLR4 / NF-kB途径抑制肾缺血再灌注损伤炎性细胞因子的释放和肾小管上皮细胞的凋亡。
机译:含微泡的miRNA-153-3p诱导近端肾小管上皮细胞凋亡,并参与肾间质纤维化
机译:激酶抑制剂-LZM缀合物的靶向肾纤维化局部治疗近端管状上皮细胞
机译:JAK2 / Y343 / STAT 5信号轴是促红细胞生成素介导的抗肾小管上皮细胞缺血性损伤的保护所必需的。
机译:血小板衍生的微泡分泌的miR-191诱导肾小管上皮细胞凋亡并通过抑制CBS参与肾缺血-再灌注损伤
机译:Lentivirus介导的SRC同源性2结构域的蛋白酪氨酸磷酸酶2通过抑制肾缺血再灌注损伤的TLR4 / NF-KB途径抑制肾小管上皮细胞的炎性细胞因子和凋亡的释放