机译:Chrysophanol通过Up-Consemate MiR-216A保护PC12细胞免受氧葡萄糖剥夺诱发的损伤
Jining 1 Peoples Hosp Dept Neurol Jining Peoples R China;
Jining 1 Peoples Hosp Dept Tradit Chinese Med Jining Peoples R China;
Jining 1 Peoples Hosp Dept Tradit Chinese Med Jining Peoples R China;
Jining 1 Peoples Hosp Dept Tradit Chinese Med Jining Peoples R China;
Jining 1 Peoples Hosp Dept Tradit Chinese Med Jining Peoples R China;
Chrysophanol; cerebral stroke; oxygen and glucose deprivation; miR-216a; PI3K; AKT and JAK2; STAT3 pathways;
机译:Chrysophanol通过Up-Consemate MiR-216A保护PC12细胞免受氧葡萄糖剥夺诱发的损伤
机译:IL-32的敲低通过NRF2 / NF-Kappa B途径的活化保护PC12细胞免受氧 - 葡萄糖剥夺/雷诺抑制的损伤
机译:IL-32的敲低通过NRF2 / NF-κB途径的激活保护PC12细胞免受氧血糖剥夺/雷诺抑制的损伤
机译:PC12细胞氧气和葡萄糖剥夺模型及HSP70蛋白的检测
机译:间充质干细胞的封装可作为中风的一种潜在治疗方法,其模型为氧葡萄糖剥夺系统。
机译:Chrysophanol通过Up-Consemate MiR-216A保护PC12细胞免受氧葡萄糖剥夺诱发的损伤
机译:Paeoniflorin通过激活JAK2 / Stat3信号传导保护PC12细胞免受氧葡萄糖剥夺/雷诺抑制的损伤