机译:突变体p53功能的增益可以是人骨骨肉瘤Mg63细胞的去分化到3ab-OS癌症干细胞的根本
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Laboratory of Biochemistry Department of Experimental Biomedicine and Clinical Neurosciences;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
Department of Molecular Medicine and Medical Biotechnology University of Naples Federico II;
Department of Molecular Medicine and Medical Biotechnology University of Naples Federico II;
Institute for Cancer Research and Molecular Medicine and Center of Biotechnology College of;
Laboratory of Biochemistry Department of Biological Chemical and Pharmaceutical Sciences and;
3AB-OS cells; Cancer cell dedifferentiation; Cancer stem cells; Human osteosarcoma; Mutant p53 gain of function;
机译:突变的p53功能获得可能是人骨肉瘤MG63细胞向3AB-OS癌症干细胞去分化的根源
机译:MicroRNA-29b-1损害人骨肉瘤3AB-OS癌症干细胞的体外细胞增殖,自我更新和化学抗药性
机译:突变的p53功能获得:通过消除突变的p53表达减少人类癌细胞系的恶性肿瘤
机译:不同的抗癌药物对人MCF-7乳腺癌细胞46位丝氨酸p53蛋白磷酸化的影响
机译:使用重组突变体/野生型p53杂合性人类乳腺上皮细胞培养模型确定p53突变在人类乳腺癌进展中的作用。
机译:MicroRNA-29b-1损害人骨肉瘤3AB-OS癌症干细胞的体外细胞增殖自我更新和化学抗药性
机译:突变的p53功能获得可能是人骨肉瘤MG63细胞向3AB-OS癌症干细胞去分化的根源。