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机译:异喹啉改善缺氧/ reoxygenation诱导的H9C2心肌细胞凋亡通过线粒体依赖性途径
Department of Cardiology Shanghai General Hospital of Nanjing Medical University;
Department of Cardiology Shanghai General Hospital of Nanjing Medical University;
Tongji University School of Medicine;
Department of Cardiology Shanghai General Hospital of Nanjing Medical University;
Isoquercetin; Hypoxia/reoxygenation; H9C2 cells; Apoptosis; ROS generation; Mitochondrial pathway;
机译:异喹啉改善缺氧/ reoxygenation诱导的H9C2心肌细胞凋亡通过线粒体依赖性途径
机译:Clematichinenoside(AR)通过线粒体介导的信号通路减弱缺氧/复氧诱导的H9c2心肌细胞凋亡。
机译:Clematichinenoside(AR)通过线粒体介导的信号通路减弱缺氧/复氧诱导的H9c2心肌细胞凋亡。
机译:DHHP-6保护H9C2心肌细胞免受缺氧/雷诺诱导的氧化损伤
机译:卡维地洛对H9C2大鼠心肌细胞硫氧还蛋白途径的影响
机译:Clematichinenoside(AR)通过线粒体介导的信号通路减弱缺氧/复氧诱导的H9c2心肌细胞凋亡。
机译:Clematichinenoside(aR)通过线粒体介导的信号通路减弱缺氧/复氧诱导的H9c2心肌细胞凋亡