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机译:AMPA受体激活氯胺酮代谢物的无关抗抑郁作用 - 诺尔汀
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Neuronal Network Department of Basic Medical Sciences Institute of Medical Sciences;
Department of Psychiatry and Behavioral Neurobiology University of Alabama at Birmingham;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Research Headquarters Taisho Pharmaceutical Co. Ltd;
Research Headquarters Taisho Pharmaceutical Co. Ltd;
Research Headquarters Taisho Pharmaceutical Co. Ltd;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
Department of Psychiatry and Behavioral Neurobiology University of Alabama at Birmingham;
Division of Neuronal Network Department of Basic Medical Sciences Institute of Medical Sciences;
Division of Clinical Neuroscience Chiba University Center for Forensic Mental Health;
AMPAR; Antidepressant; BDNF; Esketamine; NMDAR; Norketamine;
机译:AMPA受体激活氯胺酮代谢物的无关抗抑郁作用 - 诺尔汀
机译:氯胺酮代谢物缺乏NMDA抑制作用,激活AMPA受体并具有抗抑郁作用
机译:(R)-Ketamine通过AMPA受体 - 依赖性机制诱导前额外5-HT释放比(S) - 酮和氯胺酮代谢物增加更大增加
机译:增强型AMPA受体Glur1亚基表达和脑干疼痛调节电路中的神经元激活
机译:抑郁症动物模型中氯胺酮和AMPA的抗抑郁评估:机理研究。
机译:(R)-氯胺酮通过AMPA受体独立机制诱导的前额叶5-HT释放量大于(S)-氯胺酮和氯胺酮代谢物
机译:NMDAR抑制氯胺酮代谢物的无关抗抑郁药物