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机译:降低的胆碱能基础前脑完整性链接新生儿并发症和成人认知缺陷后过早出生后
German Ctr Neurodegenerat Dis Rostock Gehlsheimer Str 20 D-18147 Rostock Germany;
Univ Hosp Bonn Dept Radiol Funct Neuroimaging Grp Bonn Germany;
Tech Univ Munich Klinikum Rechts Isar Neuroimaging Ctr Dept Neuroradiol Munich Germany;
Tech Univ Munich Klinikum Rechts Isar Neuroimaging Ctr Dept Neuroradiol Munich Germany;
Univ Hosp Bonn Dept Radiol Funct Neuroimaging Grp Bonn Germany;
Univ Warwick Dept Psychol Coventry W Midlands England;
Tech Univ Munich Klinikum Rechts Isar Neuroimaging Ctr Dept Neuroradiol Munich Germany;
German Ctr Neurodegenerat Dis Rostock Gehlsheimer Str 20 D-18147 Rostock Germany;
Univ Hosp Bonn Dept Neonatol Bonn Germany;
Univ Hosp Bonn Dept Radiol Funct Neuroimaging Grp Bonn Germany;
Univ Warwick Dept Psychol Coventry W Midlands England;
Tech Univ Munich Klinikum Rechts Isar Neuroimaging Ctr Dept Neuroradiol Munich Germany;
Tech Univ Munich Klinikum Rechts Isar Neuroimaging Ctr Dept Neuroradiol Munich Germany;
Intelligence; Nucleus basalis of Meynert; Perinatal stress; Preterm birth; Structural MRI; Substantia innominata;
机译:降低的胆碱能基础前脑完整性链接新生儿并发症和成人认知缺陷后过早出生后
机译:过早出生的成年人的认知缺陷与降低的基础前脑完整有关
机译:过早出生的成年人的认知赤字与降低的基础前脑完整有关
机译:通过基于神经网络的专家系统来降低早产的风险
机译:影响功能的因素:霍乱毒素B亚基激活抗原呈递细胞,并通过骨形态发生蛋白9调节神经生长因子受体阳性基底前脑胆碱能神经元的胆碱能表型。
机译:早产成人的认知缺陷与基础前脑完整性降低相关。
机译:胆碱能的基础前脑完整性降低将早产后的新生儿并发症和成人认知缺陷联系起来