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机译:“低剂量瓦巴恩刺激Na-K ATP酶<α> 1亚基与血管紧张素II型1受体刺激Na-K ATP酶<α> 1亚基·近端小管细胞”[Biochim。 Biophys。 Acta 1863/11(2016)2624-2636]
Department of Physiology University of Louisville;
Department of Physiology University of Louisville;
Department of Physiology University of Louisville;
Department of Medicine University of Louisville;
Department of Physiology University of Louisville;
Department of Medicine University of Louisville;
Department of Medicine University of Louisville;
Department of Physiology and Biophysics Howard University College of Medicine;
机译:“低剂量瓦巴恩刺激Na-K ATP酶<α> 1亚基与血管紧张素II型1受体刺激Na-K ATP酶<α> 1亚基·近端小管细胞”[Biochim。 Biophys。 Acta 1863/11(2016)2624-2636]
机译:血管紧张素II通过G蛋白激活来自肾近端小管的对哇巴因不敏感的Na + -ATPase。
机译:SER938的大鼠肾Na-K泵的磷酸化是快速血管紧张素II依赖性活性刺激和近端小管细胞的刺激
机译:低剂量哇巴因刺激肾近端小管细胞中Na-K ATPaseα1亚基与血管紧张素II 1型受体的结合。
机译:低剂量Ouabain在肾近侧小管细胞中刺激与血管紧张素II型1受体的Na K ATP酶α1亚基联合