机译:UBR5是一种新型E3泛素连接酶,涉及骨骼肌肥大和萎缩的恢复
Stem Cells Ageing and Molecular Physiology Unit Exercise Metabolism and Adaptation Research;
Department of Internal Medicine Division of Endocrinology and Metabolism Carver College of;
Stem Cells Ageing and Molecular Physiology Unit Exercise Metabolism and Adaptation Research;
Stem Cells Ageing and Molecular Physiology Unit Exercise Metabolism and Adaptation Research;
Department of Internal Medicine Division of Endocrinology and Metabolism Carver College of;
Stem Cells Ageing and Molecular Physiology Unit Exercise Metabolism and Adaptation Research;
Department of Molecular Biology and GeneticsFederal Research and Clinical Center of Physical;
Department of Molecular Biology and GeneticsFederal Research and Clinical Center of Physical;
Department of Molecular Biology and GeneticsFederal Research and Clinical Center of Physical;
Laboratory of Exercise PhysiologyInstitute of Biomedical Problems of the Russian Academy of;
Department of Molecular Biology and GeneticsFederal Research and Clinical Center of Physical;
Exercise Metabolism and Adaptation Research Group Research Institute for Sport and Exercise;
Department of Molecular Biology and GeneticsFederal Research and Clinical Center of Physical;
Exercise Metabolism and Adaptation Research Group Research Institute for Sport and Exercise;
Department of Internal Medicine Division of Endocrinology and Metabolism Carver College of;
Stem Cells Ageing and Molecular Physiology Unit Exercise Metabolism and Adaptation Research;
UBR5; EDD1; E3 Ubiquitin Ligase; Hypertrophy; muscle atrophy; muscle injury; DNA methylation; remodelling;
机译:UBR5是一种新型E3泛素连接酶,涉及骨骼肌肥大和萎缩的恢复
机译:肌肉特异性E3泛素连接酶参与癌症恶病质的肌肉萎缩:一项体内外研究
机译:苦参碱通过抑制E3泛素连接酶而改善骨骼肌萎缩,并在C2C12 myotubes和小鼠中激活Akt / mtor / FoxO3α信号传导途径
机译:鉴定参与乳腺癌发病的E3泛素连接酶的靶类似物:一种合理的药物设计方法
机译:泛素蛋白连接酶Nedd4在骨骼肌萎缩紧张敏感模型中调节Notch1
机译:IKKβ抑制对E3泛素连接酶表达和空腹诱导的骨骼肌萎缩的悖论作用
机译:UBR5是一种新型E3泛素连接酶,涉及骨骼肌肥大和萎缩的恢复