机译:线粒体功能障碍联合高钙载体导致抗氧化防御受损的抗氧化防御性抗氧化剂的选择性丧失的抗氧化剂神经元
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Cologne Excellence Cluster Cellular Stress Respon D-50931 Cologne Germany;
Univ Cologne Ctr Physiol &
Pathophysiol Inst Vegetat Physiol D-50931 Cologne Germany;
mitochondria; Parkinson's disease; selective vulnerability; aging; calcium handling; redox stress;
机译:线粒体功能障碍联合高钙载体导致抗氧化防御受损的抗氧化防御性抗氧化剂的选择性丧失的抗氧化剂神经元
机译:HPRT缺乏多巴胺能神经元的分化受损:莱希·尼汉综合征神经元功能障碍的可能机制。
机译:PARK14(D331Y)PLA2G6通过在敲门鼠模型中诱导线粒体功能障碍,ER应激,乳化物损伤和转录性失调,使Implia NIGRA多巴胺能神经元早发退变。
机译:线粒体功能障碍联合高钙载体导致抗氧化防御受损的抗氧化防御性抗氧化剂的选择性丧失的抗氧化剂神经元
机译:线粒体功能障碍联合高钙载体导致抗氧化防御受损的抗氧化防御性抗氧化剂的选择性丧失的抗氧化剂神经元
机译:黑质多巴胺能神经元的钙稳态和线粒体功能障碍