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机译:环境富集通过BDNF / TRKB信号传导在神经受伤的小鼠中提高了长期记忆损伤和异常突触可塑性
Southeast Univ Zhongda Hosp Dept Anesthesiol Med Sch Nanjing 210029 Jiangsu Peoples R China;
Nanjing Univ Jinling Hosp Dept Anesthesiol Med Sch Nanjing 210002 Jiangsu Peoples R China;
Southeast Univ Zhongda Hosp Dept Anesthesiol Med Sch Nanjing 210029 Jiangsu Peoples R China;
Nanjing Univ Jinling Hosp Dept Anesthesiol Med Sch Nanjing 210002 Jiangsu Peoples R China;
Southeast Univ Zhongda Hosp Dept Anesthesiol Med Sch Nanjing 210029 Jiangsu Peoples R China;
Nanjing Univ Jinling Hosp Dept Anesthesiol Med Sch Nanjing 210002 Jiangsu Peoples R China;
Nerve injury; Memory; Environmental enrichment; Brain-derived neurotrophic factor;
机译:环境富集通过BDNF / TRKB信号传导在神经受伤的小鼠中提高了长期记忆损伤和异常突触可塑性
机译:重复的经颅磁刺激改善了与BDNF / TRKB信号相关的小鼠的后肢卸载引起的识别记忆损伤
机译:(p-clphse)(2)调节海马BDNF / TRKB信号传导,并逆转小鼠糖尿病诱导的记忆损伤
机译:PAK信号传导在小鼠突触传递和可塑性和社会记忆中的作用
机译:丰富的环境恢复长期的力量和脊柱大小扩大恢复IL-1信号受损的小鼠的记忆功能。
机译:黄芪IV通过EGR-1介导的BDNF / TRKB信号传导途径降低了通过eGR-1介导的BDNF / TRKB信号通路来增强内存并调节海马突触可塑性