首页> 外文期刊>Antimicrobial agents and chemotherapy. >Quinolone resistance mechanisms in salmonella enterica serovars hadar, kentucky, virchow, schwarzengrund, and 4,5,12:i:-, isolated from humans in switzerland, and identification of a novel qnrd variant, qnrd2, in s. hadar
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Quinolone resistance mechanisms in salmonella enterica serovars hadar, kentucky, virchow, schwarzengrund, and 4,5,12:i:-, isolated from humans in switzerland, and identification of a novel qnrd variant, qnrd2, in s. hadar

机译:沙门氏菌肠炎沙门氏菌中的喹诺酮耐药机制有瑞士的人类分离的哈达德,肯塔基,virchow,schwarzengrund和4,5,12:i:-,并鉴定了一种新的qnrd变体qnrd2。哈达

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摘要

Human isolates of Salmonella enterica serovars Hadar, Kentucky, Virchow, Schwarzengrund, and the monophasic variant of S. Typhimurium, Salmonella enterica subsp. enterica serovar 4,5,12:i:- were examined for mutations within the quinolone resistance target genes gyrA, gyrB, parC, and parE and for plasmid-mediated resistance genes. Differences were observed among the serovars. A novel variant of qnrD, qnrD2, was detected in an S. Hadar isolate.
机译:人类分离的肠炎沙门氏菌Hadar,肯塔基州,维尔奇豪,施瓦辛格隆德和鼠伤寒沙门氏菌的单相变体,肠炎沙门氏菌亚种。检查了肠血清4,5,12:i:-的喹诺酮抗性靶基因gyrA,gyrB,parC和parE中的突变以及质粒介导的抗性基因。在血清型之间观察到差异。在S. Hadar分离物中检测到qnrD的新型变体qnrD2。

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