首页> 美国卫生研究院文献>Antimicrobial Agents and Chemotherapy >Quinolone Resistance Mechanisms in Salmonella enterica Serovars Hadar Kentucky Virchow Schwarzengrund and 4512:i:− Isolated from Humans in Switzerland and Identification of a Novel qnrD Variant qnrD2 in S. Hadar
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Quinolone Resistance Mechanisms in Salmonella enterica Serovars Hadar Kentucky Virchow Schwarzengrund and 4512:i:− Isolated from Humans in Switzerland and Identification of a Novel qnrD Variant qnrD2 in S. Hadar

机译:沙门氏菌沙门氏菌哈达尔肯塔基州维尔茨豪施瓦辛格隆德和4512:i:-的喹诺酮耐药机制从瑞士的人体内分离并在圣哈达尔中鉴定出新型qnrD变体qnrD2。

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摘要

Human isolates of Salmonella enterica serovars Hadar, Kentucky, Virchow, Schwarzengrund, and the monophasic variant of S. Typhimurium, Salmonella enterica subsp. enterica serovar 4,5,12:i:− were examined for mutations within the quinolone resistance target genes gyrA, gyrB, parC, and parE and for plasmid-mediated resistance genes. Differences were observed among the serovars. A novel variant of qnrD, qnrD2, was detected in an S. Hadar isolate.
机译:人类分离的肠炎沙门氏菌Hadar,肯塔基州,维尔茨豪,施瓦辛格隆德和鼠伤寒沙门氏菌的单相变种,肠炎沙门氏菌亚种。检查了肠杆菌血清4,5,12:i:-喹诺酮抗性靶基因gyrA,gyrB,parC和parE中的突变以及质粒介导的抗性基因。在血清型之间观察到差异。在S. Hadar分离物中检测到qnrD的新型变体qnrD2。

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