首页> 外文期刊>Journal of cardiovascular electrophysiology >Atrial Tachycardia Developing after Cavo-Tricuspid Isthmus Ablation: What is the Mechanism?
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Atrial Tachycardia Developing after Cavo-Tricuspid Isthmus Ablation: What is the Mechanism?

机译:Cavo-三尖瓣峡部消融术后房性心动过速的发生机理是什么?

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摘要

A 65-year-old man with a history of a myocardial infarction underwent a cavo-tricuspid isthmus (CTI) catheter ablation for CTI-dependent atrial flutter (AFL) (Fig. 1). He was referred for electrophysiologic testing (EPS) and catheter ablation of recurrent AFL, which developed 4 years after the CTI ablation. At the beginning of the EPS, AFL persisted. During the AFL, the 12-lead electrocardiogram exhibited a negative saw-tooth pattern in the inferior leads, positive P waves in lead VI and negative P waves in lead V6. The atrial activation sequence along the tricuspid annulus (TA) and activation time within the right atrium (RA) covering the entire tachycardia cycle length appeared identical to that during the counter-clockwise CTI-dependent AFL (Fig. 1)
机译:一名有心肌梗塞病史的65岁男性接受了CTI依赖性心房扑动(AFL)腔三尖峡部峡部(CTI)导管消融术(图1)。他被转介为复发性AFL的电生理测试(EPS)和导管消融,后者在CTI消融后4年发展。在EPS开始时,AFL坚持了下来。在AFL期间,12导联心电图在下导联中呈现负锯齿形,在VI导联中呈现正P波,在V6导联中呈现负P波。沿三尖瓣环(TA)的心房激活序列和右心房(RA)的激活时间覆盖了整个心动过速周期长度,与逆时针CTI依赖的AFL期间的序列相同(图1)。

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