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Hypertrophic cardiomyopathy with midcavitary obstruction: another substrate for ventricular tachycardia?

机译:肥厚型心肌病合并中腔阻塞:另一种心室性心动过速的底物?

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摘要

The importance of the anatomic substrate for the genesis of ventricular arrhythmias has become widely accepted. Ventricular tachycardia (VT) associated with myocardial infarction typically has a critical isthmus endocardially, near the border zone of the myocardial scar.VT associated with Chagastic cardiomyopathy was the first arrhythmia described to have a predominantly epicardial substrate. Monomor-phic VT associated with dilated nonischemic cardiomyopathy, previously considered uncommon, has now been recognized to originate from basal periannular left ventricular (LV) low-voltage zone, often with a mid-myocardial or epicardial exit. Arrhythmogenic right ventricular cardiomyopathy has been associated with a periannular right ventricular substrate, with a larger region of epicardial compared with endocardial scar.
机译:解剖学基质对于心律失常的发生的重要性已被广泛接受。与心肌梗塞相关的室性心动过速(VT)通常在心肌疤痕边界区域附近的心内膜有严重的峡部。与以前认为不常见的扩张型非缺血性心肌病相关的单形室速现已被认为起源于基底环周左心室(LV)低压区,通常伴有心肌中部或心外膜出口。与心内膜瘢痕相比,致心律失常性右心室心肌病与瓣膜周围右心室基质相关,心外膜区域更大。

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