Adenosine triphosphate terminates bidirectional ventricular tachycardia in a patient with catecholaminergic polymorphic ventricular tachycardia.

摘要

Catecholammergic polymorphic ventricular tachycardia (CPVT) was first described as a distinct clinical entity by Leenhardt et al, and is characterized by the development of bidirectional ventricular tachycardia (VT), polymorphic ventricular tachycardia (PVT), or ventricular fibrillation (VF) induced by exercise or emotional stress, usually in children and young adults.1'2 The underlying cellular mechanism of these unique arrhythmias is thought to be triggered activity caused by delayed afterdepolarization. Adenosine and adenosine triphosphate are useful in treating VT caused by triggered activity in conditions other than CPVT. Here, we report the effectiveness of adenosine triphosphate in terminating bidirectional VT in a patient with CPVT.