机译:Pard3A的Siah调节在萌发区出口期间控制神经元细胞粘附。
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA,Department of Biological Sciences, University of Alberta, Edmonton, Alberta T6G 2E9, Canada;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA,Department of Physiology, Development and Neuroscience, Anatomy Building, University of Cambridge,Downing Street Cambridge CB2 3DY, UK;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA;
Department of Developmental Neurobiology, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis,TN 38105, USA;
机译:SiaH2与发芽区占用链接神经元祖细胞发生的丝粉膜和细胞外基质信号
机译:氧气张力和VHL-HIF1α路径确定神经元偏振和小脑发芽区出口的发作
机译:Zeb1通过间充质-上皮样过渡控制神经元分化和生发区退出
机译:活神经细胞神经细胞粘附分子的光学捕获和组装
机译:肝激酶B1通过调节活化的B细胞的核因子κ轻链增强剂来控制T卵泡辅助细胞的分化和生殖中心的形成。
机译:pard3a的sIaH调节控制生发区退出期间神经细胞粘附
机译:PARD3A的SIAH调节在发芽区出口期间控制神经元细胞粘附