机译:VEGF-B对于血管生长是必不可少的,但对它们的生存至关重要,而VEGF-B的靶向抑制病理性血管生成
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
Ludwig Institute for Cancer Research, Uppsala University, SE-751 24 Uppsala, Sweden;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
Neurodegeneration Labs, Protein Chemistry, Genentech, Inc. San Francisco, CA 94080;
CSL Limited, Parkville, Victoria 3052, Australia;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
Department of Biotechnology and Molecular Medicine, University of Kuopio, FIN-70211, Kuopio, Finland;
Department of Radiology, Medical Imaging Centre of the Affiliated Hospital, Weifang Medical University, Weifang 261042, China;
Department of Physiology, Kinki University School of Medicine, Osaka 589-8511, Japan;
Sanofi-Aventis Recherche, Cardiovascular-Thrombosis Research Department, 31036 Toulouse Cedex, France;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
TRIAD Technology Center, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
Center for Vascular Biology, University of Connecticut, Farmington, CT 06030;
Department of Biochemistry and Medical Genetics, University of Manitoba, Winnipeg, Canada MB R3E 3P5;
Buck Institute for Age Research, Novato, CA 94945;
TRIAD Technology Center, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224;
Sanofi-Aventis Recherche, Cardiovascular-Thrombosis Research Department, 31036 Toulouse Cedex, France;
CSL Limited, Parkville, Victoria 3052, Australia;
Department of Biotechnology and Molecular Medicine, University of Kuopio, FIN-70211, Kuopio, Finland;
Department of Microbiology, Karolinska Institute, 171 77 Stockholm, Sweden;
Neurodegeneration Labs, Protein Chemistry, Genentech, Inc. San Francisco, CA 94080;
National Eye Institute, National Institutes of Health, Bethesda, MD 20892;
apoptosis; vascular survival; ocular neovascularization;
机译:再现VEGF-B的α1螺旋的循环肽与VEGFR-1和VEGFR-2结合并抑制血管生成和肿瘤生长
机译:模拟VEGF-A和VEGF-B结合位点的肽可抑制VEGFR-1 / -2驱动的血管生成,肿瘤生长和转移
机译:在帕金森氏病的培养模型中,血管内皮生长因子B(VEGF-B)上调且外源性VEGF-B具有神经保护作用
机译:胶质母细胞瘤生长的耦合数学模型,预先存在的血管互联,血管生成和血液灌注
机译:SLIT2-ROBO信号通路作为衰减病理糖尿病肾小球血管生成的潜在目标
机译:VEGF-B对于血管生长是必不可少的但对它们的生存至关重要而VEGF-B的靶向抑制病理性血管生成
机译:VEGF-B对于血管生长是必不可少的,但对它们的生存至关重要,而VEGF-B的靶向抑制病理性血管生成