机译:疟疾引发由于干扰素-γ诱导的Toll样受体表达和功能增强而引起的先天免疫应答
lmmunopathology Laboratory, Rene Rachou Institute, Fundaqao Oswaldo Cruz, Avenue Augusto de Lima 1715, MG 30190, Belo Horizonte, Brazil Department of Medicine, Division of Infectious Diseases and Immunology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605;
Department of Medicine, Division of Infectious Diseases and Immunology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605;
lmmunopathology Laboratory, Rene Rachou Institute, Fundaqao Oswaldo Cruz, Avenue Augusto de Lima 1715, MG 30190, Belo Horizonte, Brazil;
Department of Medicine, Division of Infectious Diseases and Immunology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605;
lmmunopathology Laboratory, Rene Rachou Institute, Fundaqao Oswaldo Cruz, Avenue Augusto de Lima 1715, MG 30190, Belo Horizonte, Brazil;
Department of Medicine, Division of Infectious Diseases and Immunology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605;
Centro de Pesquisas em Medicina Tropical de Rondonia, 364, Km 4.5, RO 78900, Porto Velho, Brazil;
Centro de Pesquisas em Medicina Tropical de Rondonia, 364, Km 4.5, RO 78900, Porto Velho, Brazil;
Centro de Pesquisas em Medicina Tropical de Rondonia, 364, Km 4.5, RO 78900, Porto Velho, Brazil;
Centro de Pesquisas em Medicina Tropical de Rondonia, 364, Km 4.5, RO 78900, Porto Velho, Brazil;
Experimental Cardiovascular Research, Lund University, SE-205 02 Malmoe, Sweden;
lmmunopathology Laboratory, Rene Rachou Institute, Fundaqao Oswaldo Cruz, Avenue Augusto de Lima 1715, MG 30190, Belo Horizonte, Brazil Department of Medicine, Division of Infectious Diseases and Immunology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605;
lmmunopathology Laboratory, Rene Rachou Institute, Fundaqao Oswaldo Cruz, Avenue Augusto de Lima 1715, MG 30190, Belo Horizonte, Brazil Department of Medicine, Division of Infectious Diseases a;
机译:孕酮通过抑制NF-κB活化并增强SOCS1表达来抑制巨噬细胞中Toll样受体4介导的先天免疫应答。
机译:疟原虫具有免疫惰性,但通过向Toll样受体9呈递疟疾DNA可以从根本上增强先天应答。
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机译:在先天免疫应答中的可收费性受体信号传导途径的调节
机译:确定Toll样受体在对hermesii疏螺旋体和其他非T细胞体液免疫抗原靶点的先天性和适应性免疫反应中的作用
机译:由于干扰素-γ诱导的Toll样受体表达和功能增强疟疾引发了先天免疫应答
机译:由于干扰素-γ诱导的Toll样受体表达和功能增强,疟疾引发了先天免疫应答