机译:内源性芳基烃受体配体作用于树突状细胞和T细胞,以抑制实验性自身免疫性脑脊髓炎
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
Center for Neurologic Diseases, The Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02115;
机译:芳烃受体配体作用于树突状细胞和T细胞,以抑制变应性鼻炎患者的Th17反应
机译:芳烃受体配体作用于树突状细胞和T细胞,以抑制变应性鼻炎患者的Th17反应
机译:皮肤的UV-B照射通过在树突细胞中的芳基烃受体信号来改善实验性自身免疫性脑炎
机译:U937细胞和人类巨噬细胞中的ARYL碳氢化合物受体/二恶英受体
机译:激素和树突状细胞:对自身免疫性疾病实验性自身免疫性脑脊髓炎的发作的影响。
机译:从封面开始:内源性芳基烃受体配体作用于树突状细胞和T细胞以抑制实验性自身免疫性脑脊髓炎
机译:内源性芳基烃受体配体作用于树突状细胞和T细胞,以抑制实验性自身免疫性脑脊髓炎