机译:MYC原癌基因与抑制Aurora-B激酶之间合成致死性相互作用的治疗潜力
Department of Microbiology and Immunology, G. W. Hooper Research Foundation, University of California, San Francisco, CA 94143;
rnDepartment of Microbiology and Immunology, G. W. Hooper Research Foundation, University of California, San Francisco, CA 94143;
rnDivision of Hematology/Oncology, Department of Medicine, University of California, San Francisco, CA 94143;
rnDepartment of Basic Medical Sciences, College of Medicine, University of Arizona, Phoenix, AZ 85004;
rnDepartment of Microbiology and Immunology, G. W. Hooper Research Foundation, University of California, San Francisco, CA 94143;
rnDepartment of Microbiology and Immunology, G. W. Hooper Research Foundation, University of California, San Francisco, CA 94143;
apoptosis; aurora kinase; autophagy synthetic lethality; targeted therapy; chromosomal passenger protein complex;
机译:抑制Aurora激酶与化疗组合诱导合成致死率,克服Myc过表达淋巴瘤中的化学性
机译:Polo样激酶1作为合成致死诱导选择性靶向BRCA1缺陷癌细胞的治疗方法
机译:抑制BRCT(BRCA1)-磷酸蛋白相互作用增强olaparib在乳腺癌细胞中的细胞毒性作用:合成致死性治疗方案的概念研究证明(第134卷,第511页,2012年)
机译:KRAS与Cdk4之间的合成致死相互作用的混合胶束siRNA递送系统,用于基因特异性癌症治疗。
机译:通过研究与MYC解除管制的合成致死相互作用,深入了解MYC生物学。
机译:MYC原癌基因与抑制Aurora-B激酶之间合成致死性相互作用的治疗潜力
机译:MYC原癌基因与抑制Aurora-B激酶之间合成致死性相互作用的治疗潜力