机译:HMGB1与Toll样受体4结合并激活巨噬细胞因子释放需要关键半胱氨酸
Laboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnDepartments of Medicine and Children's Health, Karolinska Institute, 17176 Stockholm, Sweden;
rnDepartments of Women's and Children's Health, Karolinska Institute, 17176 Stockholm, Sweden;
rnDepartment of Emergency Medicine, North Shore University Hospital, Manhasset, NY 11030;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnDepartment of Medicinal Chemistry, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030;
rnLaboratory of Host Defense, WPI Immunology Frontier Research Center, Osaka University, Osaka 565-0871, Japan;
rnDepartment of Medicine and Clinical Chemistry, University of Heidelberg, 69120 Heidelberg, Germany;
rnDepartments of Medicine and Children's Health, Karolinska Institute, 17176 Stockholm, Sweden;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030 Departments of Women's and Children's Health, Karolinska Institute, 17176 Stockholm, Sweden;
rnLaboratory of Biomedical Science, The Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030 Departments of Women's and Children's Health, Karolinska Institute, 17176 Stockholm, Sweden;
innate immunity; infection; sterile inflammation; TNF; proximity ligation; assay;
机译:在表达人而非鼠类Toll样受体4-MD-2-CD14的细胞中,健壮的NF-κB活化和促炎细胞因子的释放需要用葡萄糖胺代替百日咳博德特氏菌脂质A磷酸基团。
机译:氧化还原活性痕量金属诱导的高迁移率组箱1(HMGB1)和成纤维细胞样滑膜细胞中的炎性细胞因子是依赖于损伤的受体4(TLR4)
机译:Toll样受体在巨噬细胞HMGB1释放中的作用
机译:通过Fceri和Corl样受体对肥大细胞的促成显着增加了炎症细胞因子的产生
机译:Toll样受体4活化巨噬细胞中磷脂酸磷酸水解酶1和IVA组磷脂酶A2的功能,激活和抑制。
机译:HMGB1与Toll样受体4结合并激活巨噬细胞因子释放需要关键半胱氨酸
机译:HMGB1与Toll样受体4结合并激活巨噬细胞因子释放需要关键半胱氨酸