机译:酪蛋白激酶2通过磷酸化和降解肌醇六磷酸激酶2介导细胞存活。
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205;
The Solomon H. Snyder Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, MD 21205,Department of Psychiatry and Behavioral Sciences, Johns Hopkins University School of Medicine, Baltimore, MD 21205,Department of Pharmacology and Molecular Sciences,Johns Hopkins University School of Medicine, Baltimore, MD 21205;
机译:HSP90通过肌醇六磷酸激酶-2调节细胞存活
机译:细菌颗粒细胞中肌醇六磷酸激酶-2通过蛋白质4.1n调节幼儿群细胞和电机协调
机译:p53介导的凋亡需要肌醇六磷酸激酶2
机译:硫代磷酸含溶质通过内在途径介导人乳腺腺癌MCF-7细胞凋亡
机译:肌醇六磷酸激酶-2对p53介导的细胞凋亡的调节
机译:酪蛋白激酶2通过磷酸化和降解肌醇六磷酸激酶2介导细胞存活。
机译:酪蛋白激酶2通过磷酸化和降解肌醇六磷酸激酶2介导细胞存活。