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机译:IL-38与IL-36受体结合,对免疫细胞具有类似于IL-36受体拮抗剂的生物学作用
Department of Medicine, University of Colorado Denver, Aurora, CO 80045,Department of Medicine, University Medical Center Nijmegen, 6500 HB,Nijmegen, The Netherlands;
R&D Systems, Inc., Minneapolis, MN 55413;
R&D Systems, Inc., Minneapolis, MN 55413;
R&D Systems, Inc., Minneapolis, MN 55413;
Department of Medicine, University of Colorado Denver, Aurora, CO 80045;
Department of Medicine, University Medical Center Nijmegen, 6500 HB,Nijmegen, The Netherlands;
Department of Medicine, University Medical Center Nijmegen, 6500 HB,Nijmegen, The Netherlands;
Department of Medicine, University Medical Center Nijmegen, 6500 HB,Nijmegen, The Netherlands;
R&D Systems, Inc., Minneapolis, MN 55413;
R&D Systems, Inc., Minneapolis, MN 55413;
Department of Medicine, University of Colorado Denver, Aurora, CO 80045,Department of Medicine, University Medical Center Nijmegen, 6500 HB,Nijmegen, The Netherlands;
inflammation; IL-1F10; IL-1F5; IL-1Rrp2;
机译:IL-36受体拮抗剂,特别是IL-38
机译:IL-36受体拮抗剂缺乏导致由于过度招募免疫细胞而导致伤口愈合
机译:IL-36受体拮抗抗体抑制临床前模型皮炎的临床前症的炎症反应
机译:新型糖皮质激素拮抗剂组织组织拮抗剂组织术的差异效应和ru486(MIFEPRISTONE)对ATT20细胞系糖皮质激素的糖皮质激素核易位
机译:优化嵌合抗原受体(汽车)T细胞通过IL-36γ进行增强和扩大的抗肿瘤免疫应答
机译:IL-38与IL-36受体结合对免疫细胞具有类似于IL-36受体拮抗剂的生物学作用
机译:SARS-COV-2感染诱导因突变导致IL-36受体拮抗剂的激活切割位点导致的白细胞介素三十六(IL-36)受体拮抗剂(DITRA)的严重爆发缺乏缺陷