机译:动脉粥样硬化的几何形状以von Willebrand因子依赖性方式加剧病理性血栓形成后狭窄
Department of Biochemistry, Cardiovascular Research Institute Maastricht, Maastricht University, 6200 MD Maastricht, The Netherlands,Department of Atherothrombosis and Vascular Biology, Baker International Diabetes Institute, Heart and Diabetes Institute, Melbourne, Victoria 8008, Australia;
BIOS/Lab on a Chip Group, MESA+ Institute for Nanotechnology, University of Twente, 7500 AE Enschede, The Netherlands;
Department of Biochemistry, Cardiovascular Research Institute Maastricht, Maastricht University, 6200 MD Maastricht, The Netherlands;
BIOS/Lab on a Chip Group, MESA+ Institute for Nanotechnology, University of Twente, 7500 AE Enschede, The Netherlands;
BIOS/Lab on a Chip Group, MESA+ Institute for Nanotechnology, University of Twente, 7500 AE Enschede, The Netherlands;
Department of Biochemistry, Cardiovascular Research Institute Maastricht, Maastricht University, 6200 MD Maastricht, The Netherlands;
机译:剪切速率梯度促进弱粘合蛋白的双相血栓形成,例如von Willebrand因子依赖性方式的纤维蛋白原
机译:全血流条件下von Willebrand因子依赖性血栓形成组织因子的功能性
机译:在表达von Willebrand因子变体的von Willebrand因子缺陷型小鼠中,血栓形成发生改变,与胶原蛋白或GPIIbIIIa结合不良。
机译:von Willebrand因子A1域中IIb型突变诱导的构象变化的动力学
机译:蛇毒凝集素(VCA)。蛇毒的血小板聚集原理:冯·威勒伯兰德依赖血小板的聚集。
机译:动脉粥样硬化的几何形状以von Willebrand因子依赖性方式加剧病理性血栓形成后狭窄
机译:动脉粥样硬化的几何形状以von Willebrand因子依赖性方式加剧病理性血栓形成后狭窄