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DO WE EVER STOP LEARNING?

机译:我们曾经停止学习吗?

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SINCE AT LEAST THE 1500s, THE ADAGE "You can't teach an old dog new tricks" has preached the impossibility of schooling older folks. The trope still manages to color stereotypes of aging as more of a downhill slide than a journey toward wisdom. But 16th-century know-it-alls didn't have access to 21st-century neuroscience, and a growing body of research suggests that late-in-life learning is likelier than Renaissance pundits could ever have imagined. In fact, education does an aging noggin good. Our brains are bafflingly complex at any age. The average adult has around 86 billion neurons, connected by synapses-tiny gaps where these cells exchange chemical signals. Each head hums with hundreds of trillions of these connections, all sending and receiving tiny bits of information and instructions. During the 20th century, imaging tools like MRIs and BEGs finally let neurologists examine how those paths change as our minds mature, and they revealed that ageist notions of doddering seniors were quite mistaken. Throughout life, our noggins constantly rewire themselves. Some scientists suspected as much as far back as the late 1800s. But it wasn't until the late 1960s, when British neuroscien-tist Geoffrey Raisman spied growth in damaged cerebral regions of rats through an electron microscope, that anyone managed to catch them forging new connections-an ability called neuro-plasticity. "Molecular changes occur each time we learn something new," says Kaitlin Casaletto, a neuropsychologist at the Memory and Aging Center at the University of California, San Francisco (UCSF). As we encounter novel information, our brains release chemicals that subtly alter our synapses and change the organ's physical form by blazing new neural pathways. Such tweaks stop only with degenerative disease or death.
机译:由于至少1500岁,格言“你不能教一只老狗的新技巧”已经讲道了学校老年人的不可能。该牵引仍然可以管理老化的刻板印象,因为比智慧的旅程更多的下坡滑梯。但是,16世纪的诀窍都无法获得21世纪的神经科学,而且越来越多的研究体系表明,生命晚期学习比文艺复兴的专家能够想象。事实上,教育做了一个老化的noggin好。在任何年龄的年龄,我们的大脑都会变得困扰。普通成年人有大约860亿神经元,通过突触 - 微小的空隙连接,其中这些细胞交换化学信号。每个头部嗡嗡声都有数百万亿的连接,所有这些连接都发送和接收了微小的信息和说明。在20世纪,在20世纪,像MRIS这样的成像工具最终让神经科学家审查这些道路如何随着我们的思想成熟而变化,并且他们透露了散发式老年人的年龄识别概念被误解了。整个生命中,我们的诺吉斯不断地重新围绕自己。一些科学家们在1800年代后期被追溯到遥远。但直到20世纪60年代末,当英国神经照片 - TIST Geoffrey Raisman通过电子显微镜进行损坏的大鼠脑区的增长,任何人都会抓住它们锻造新的联系 - 一种称为神经可塑性的能力。 “每次我们学习新事物时都会发生分子变化,”旧金山大学旧金山(UCSF)内存和老化中心的神经心理学家Kaitlin Casaletto说。正如我们遇到新颖的信息,我们的大脑释放化学物质,这些化学品可以通过燃烧新的神经途径来改变我们的突触并改变器官的物理形式。这种调整仅次于退行性疾病或死亡。

著录项

  • 来源
    《Popular Science》 |2020年第4期|26-27|共2页
  • 作者

    ERIN BLAKEMORE;

  • 作者单位
  • 收录信息 美国《科学引文索引》(SCI);美国《工程索引》(EI);美国《生物学医学文摘》(MEDLINE);美国《化学文摘》(CA);
  • 原文格式 PDF
  • 正文语种 eng
  • 中图分类
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  • 入库时间 2022-08-18 21:04:17

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