...
机译:Cyanidin-3-葡萄糖苷逆转乙醇诱导的神经突增生抑制:糖原合酶激酶3 Beta的作用。
Department of Internal Medicine University of Kentucky College of Medicine 124C Combs Research Building 800 Rose Street Lexington KY 40536 USA;
Department of Internal Medicine University of Kentucky College of Medicine 124C Combs Research Building 800 Rose Street Lexington KY 40536 USA;
Department of Internal Medicine University of Kentucky College of Medicine 124C Combs Research Building 800 Rose Street Lexington KY 40536 USA;
National Institute for Occupational Safety and Health Morgantown WV 26505 USA;
Graduate Center for Toxicology University of Kentucky 232 Bosomworth Lexington KY 40536 USA;
Institute for Nutritional Sciences Shanghai Institutes for Biological Sciences Chinese Academy of Sciences Shanghai 200031 People’s Republic of China;
Department of Internal Medicine University of Kentucky College of Medicine 124C Combs Research Building 800 Rose Street Lexington KY 40536 USA;
Alcohol; Development; Differentiation; Fetal alcohol syndrome; Neuroprotection;
机译:Cyanidin-3-glucoside逆转乙醇诱导的神经突增生抑制:糖原合酶激酶3 Beta的作用。
机译:链脲佐菌素通过Pi3k-akt和糖原合成酶激酶3β在神经2a细胞中诱导神经沸石过多
机译:糖原合酶激酶3和Axin在β-catenin非依赖性途径中调节神经母细胞瘤细胞的神经突向外生长。
机译:Paullones对糖原合酶激酶3抑制作用建模不确定性的模糊处理
机译:丝氨酸-苏氨酸激酶糖原合酶激酶-3β和蛋白激酶CK2在乳腺肿瘤发生和上皮向间质转化中的作用。
机译:Cyanidin-3-葡萄糖苷逆转乙醇诱导的神经突生长的抑制作用:糖原合酶激酶3β的作用
机译:Cyanidin-3-葡萄糖苷逆转乙醇诱导的神经突生长的抑制作用:糖原合酶激酶3β的作用