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机译:短暂性前脑缺血后沙土鼠海马CA1区过氧化的过氧化物酶和甘油醛-3-磷酸脱氢酶的免疫反应性和蛋白质水平发生了变化。
Department of Anatomy and Neurobiology College of Medicine Hallym University Chuncheon 200-702 South Korea;
Department of Anatomy and Neurobiology College of Medicine Hallym University Chuncheon 200-702 South Korea;
Department of Biomedical Science and Research Institute for Bioscience and Biotechnology Hallym University Chuncheon 200-702 South Korea;
Department of Anatomy and Cell Biology College of Veterinary Medicine and BK21 Program for Veterinary Science Seoul National University Seoul 151-742 South Korea;
Department of Anatomy and Cell Biology College of Veterinary Medicine and BK21 Program for Veterinary Science Seoul National University Seoul 151-742 South Korea;
Department of Anatomy and Neurobiology College of Medicine Hallym University Chuncheon 200-702 South Korea;
Peroxiredoxins; Glyceraldehyde-3-phosphate dehydrogenase; Hyperoxidation; Reactive oxygen species; Cerebral ischemia;
机译:短暂性前脑缺血后,沙土鼠海马CA1区的过氧化过氧化物酶和3-磷酸甘油醛脱氢酶的免疫反应性和蛋白质水平发生变化。
机译:沙土鼠短暂前脑缺血后,海马CA1区的I型腺苷酸环化酶的免疫反应性和活性发生了变化。
机译:短暂性脑缺血后沙鼠海马CA1区神经元和星形胶质细胞中蛋白质二硫键异构酶的免疫反应性和蛋白质水平的变化。
机译:FK506促进了蛋白激酶术后缺血性扰动的标准化,并在Gerbil海马CA1部门中抑制蛋白激酶和酪氨酸磷酸化
机译:大鼠的短暂性前脑缺血会触发易感性和耐药性脑区域蛋白质磷酸化和丙酮酸脱氢酶活性的变化。
机译:缺血预处理可在短暂性脑缺血后维持沙鼠海马CA1区神经元中葡萄糖激酶和葡萄糖激酶调节蛋白的免疫反应性
机译:短暂性前脑缺血后沙鼠海马Ca1区铁,铁蛋白和转铁蛋白的时间和细胞类型特异性变化