机译:高p-catenin / Tcf-4活性通过直接调节AKT2基因表达赋予神经胶质瘤进展
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University,Nanjing 210029;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University,Nanjing 210029;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University,Nanjing 210029;
Department of Neurosurgery, Tiantan Hospital, Capital Medical University, Beijing 100050, China;
Department of Neurosurgery, The First Affiliated Hospital of Nanjing Medical University,Nanjing 210029;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
Department of Neurosurgery, Tianjin Medical University General Hospital, Laboratory of Neuro-Oncology,Tianjin Neurological Institute, Key Laboratory of Neurotrauma, Variation and Regeneration, Ministry of Education and Tianjin Municipal Government, Tianjin 300052, China;
glioma; 3-catenin; tcf-4; akt2.;
机译:肿瘤分级相关的神经球蛋白表达受PPARγ负调节,并赋予神经胶质瘤进展抗氧化活性
机译:TIMP3差异表达通过调节方向性持久性内皮细胞迁移影响黑素瘤的肿瘤进展和血管生成
机译:槲皮素和乙醇减弱LDLR-/-小鼠中对氧磷合酶1(PON1)基因表达和PON1活性的同时上调与动脉粥样硬化斑块的进展。
机译:人胶质瘤的基因表达谱
机译:C6胶质瘤细胞中大鼠星形胶质细胞中谷氨酰胺合成酶基因表达的调控及其与磷酸甘油脱氢酶的差异调控。
机译:高β-catenin/ Tcf-4活性通过直接调节AKT2基因表达赋予神经胶质瘤进展
机译:高β-catenin/ Tcf-4活性通过直接调节AKT2基因表达赋予神经胶质瘤进展