机译:对抗微管蛋白化学疗法的敏感性由MCL1和FBW7调节
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
The Walter and Eliza Hall Institute of Medical Research, Parkville, Victoria 3052, Australia;
Department of Protein Chemistry, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA,Department of Protein Chemistry, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Bioinformatics, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Bioinformatics, Genentech, South San Francisco, California 94080, USA;
Department of Physiological Chemistry, Genentech, South San Francisco, California 94080, USA;
Department of Molecular Biology, Genentech, South San Francisco, California 94080, USA;
Department of Biochemical Pharmacology, Genentech, South San Francisco, California 94080, USA;
Department of Biochemical Pharmacology, Genentech, South San Francisco, California 94080, USA;
Department of Structural Biology, Abbott Laboratories, Abbott Park, Illinois 60064, USA;
Department of Protein Chemistry, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
Department of Molecular Biology, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
Department of Early Discovery Biochemistry, Genentech, South San Francisco, California 94080, USA;
Department of Research Oncology, Genentech, South San Francisco, California 94080, USA;
The Walter and Eliza Hall Institute of Medical Research, Parkville, Victoria 3052, Australia,Department of Medical Biology, University of Melbourne, Parkville, Victoria 3010, Australia;
Department of Physiological Chemistry, Genentech, South San Francisco, California 94080, USA;
机译:PTBP1对MCL1表达的调节调节抗微管蛋白化学疗法诱导的细胞凋亡
机译:SCF〜(FBW7)通过靶向MCL1进行泛素化和破坏来调节细胞凋亡
机译:对抗胰岛素化学治疗剂的敏感性由可光活化的纳米脂质体增强
机译:PTEN基因抑制食管鳞状细胞癌的细胞增殖并提高化疗药物的敏感性
机译:靶向MEK5,MEK1 / 2和/或PI3K途径将上皮细胞转型的上皮性反转,并通过间充质表型提高乳腺癌和脑癌中的化疗敏感性
机译:PTBP1调节MCL1表达可调节抗微管蛋白化学疗法诱导的细胞凋亡
机译:DNA甲基化调节的miR-155-5p通过抑制MAP3K10抑制食管癌细胞对辐射和多种化学治疗药物的敏感性