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机译:炎性体介导的营养不良调节NAFLD和肥胖的发展
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA,Department of Cell Biology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Pathology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Center for Genome Sciences and Systems Biology, Washington University School of Medicine, St Louis, Missouri 63108, USA,Division of Allergy and Immunology, Department of Internal Medicine, Washington University School of Medicine, St Louis, Missouri 63108, USA;
Department of Laboratory Medicine, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut 06520, USA;
Department of Internal Medicine, Yale University School of Medicine, New Haven, Connecticut 06520, USA,Howard Hughes Medical Institute, Chevy Chase, Maryland 20815, USA;
Center for Genome Sciences and Systems Biology, Washington University School of Medicine, St Louis, Missouri 63108, USA;
Department of Pediatrics, Rady Children's Hospital San Diego, University of California at San Diego, LaJolla, California 92093, USA;
Department of Immunobiology, Yale University School of Medicine, New Haven, Connecticut 06520, USA,Howard Hughes Medical Institute, Chevy Chase, Maryland 20815, USA;
机译:肝脏GDF15受展开的蛋白质反应的CHOP调节,并在肥胖小鼠中缓解NAFLD进展
机译:微生物群:由炎性体缺乏症驱动的营养不良会加剧肝脂肪变性并控制NAFLD进展的速度
机译:难治性肥胖,肥胖和肥胖
机译:肥胖青少年肠道菌群的营养不良
机译:调查Plin2-Null小鼠抗肥胖和NAFLD的分子途径
机译:炎性体介导的生态失调调节NaFLD和肥胖的进展
机译:NAFLD进化与血浆促氧化生物标志物相关,而不是肝组织测量的氮代谢生物标志物,肥胖症和代谢综合征