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机译:组蛋白肽H471–94 sub>在控制狼疮方面比肽表位鸡尾酒更有效:免疫调节机制
Division of Rheumatology, Departments of Medicine, Northwestern University Feinberg School of Medicine, 240 East Huron St. McGaw # M300, Chicago, IL, 60611, USA;
Division of Rheumatology, Departments of Medicine, Northwestern University Feinberg School of Medicine, 240 East Huron St. McGaw # M300, Chicago, IL, 60611, USA;
Division of Rheumatology, Departments of Medicine, Northwestern University Feinberg School of Medicine, 240 East Huron St. McGaw # M300, Chicago, IL, 60611, USA;
Division of Rheumatology, Departments of Medicine, Northwestern University Feinberg School of Medicine, 240 East Huron St. McGaw # M300, Chicago, IL, 60611, USA;
Division of Rheumatology, Departments of Medicine, Northwestern University Feinberg School of Medicine, 240 East Huron St. McGaw # M300, Chicago, IL, 60611, USA;
Systemic lupus erythematosus; T cells; DC; peptides; tolerance; autoimmunity;
机译:单独的组蛋白肽H4 71-94在控制狼疮方面比肽表位混合物更有效:免疫调节机制。
机译:核小体组蛋白肽表位诱导的调节免疫可有效抑制人狼疮的主要致病步骤
机译:铜与组蛋白H2B的32-62和94-125肽片段的有效结合
机译:通过化学选择腙形成同时对多表位肽鸡尾酒的脂质
机译:抗菌II类细菌素中肽-肽和肽-表面相互作用的机制。
机译:单独的组蛋白肽H471-94比控制狼疮的肽表位的鸡尾酒更有效:免疫调节机制
机译:利用核心的耐含致素组蛋白肽表位,用于选择性下调狼疮(过去,礼物和未来)的病原自身免疫反应