机译:Cx36使通道耦合人胰岛β细胞,并与胰岛素表达相关
1Surgical Research Unit, Department of Surgery 2Cell Isolation and Transplantation Center, Department of Surgery, Geneva University Hospitals, Geneva, Switzerland 3Department of Cell Physiology and Metabolism, University of Geneva School of Medicine, CMU 1, rue Michel-Servet, 1211 Geneva 4, CH, Switzerland 4Islet Research Laboratory, The Whittier Institute for Diabetes, University of California San Diego, La Jolla, CA, USA 5Department of Cell Biology, University of Groningen, Groningen, The Netherlands 6Department of Endocrinology and Metabolism, University of Pisa, Pisa, Italy 7Department of Surgery, University of Pennsylvania, Philadelphia, PA, USA;
机译:Cx36使通道耦合人胰岛β细胞,并与胰岛素表达相关。
机译:减少Cx36缝隙耦合可补偿过度活跃的K_(ATP)通道,以恢复胰岛素分泌并预防新生儿糖尿病小鼠模型中的高血糖症
机译:胰岛素样生长因子-1通过R型Ca2 +通道增加人原发性神经内分泌胰腺肿瘤细胞和胰腺神经内分泌肿瘤细胞系(BON-1)的细胞内钙浓度,并调节嗜铬粒蛋白的分泌
机译:形成的细胞团调节葡萄糖刺激的胰岛素基因表达并促进胰腺β细胞中胰岛素的分泌
机译:T型钙通道在高糖血症下胰腺β细胞的基础[钙(2 +)](i)和胰岛素分泌中的作用。
机译:Cx36使通道耦合人胰岛β细胞并与胰岛素表达相关
机译:Cx36使通道耦合人胰岛β细胞,并与胰岛素表达相关